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The BRAF (V600E) mutation analysis is predictive for anti-EGFR therapies (i.e., cetuximab and panitumumab). BRAF mutations account for approximately 12-15% of colorectal cancer patients.
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A large body of experimental and clinical work supports the view that EGFR is a relevant target for cancer therapy. The development of selective tyrosine kinase inhibitors has become an important area of drug discovery for the treatment of a variety of solid tumors such as breast, ovarian and colorectal cancers, NSCLC, and carcinoma of the head and neck.
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V-raf murine sarcoma viral oncogene homolog B1, also known as BRAF, is a protein which in humans is encoded by the BRAF gene. The BRAF gene makes a protein called B-RAF, which is involved in sending signals in cells and in cell growth. This gene may be mutated (changed) in many types of cancer, which causes a change in the B-RAF protein. This can increase the growth and spread of cancer cells.
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Genetic cancer testing is an innovative breakthrough in cancer research. It is gaining popularity worldwide and will provide answers that doctors have long been waiting for. Testing genetically will provide optimal results in cancer research.
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